Comment on: Variables associated subclinical atherosclerosis among rheumatoid arthritis patients of Gulf Cooperative Council countries

References

1. ↵
   1. Hannawi S,
   2. Hannawi H,
   3. Alokaily F,
   4. Al Salmi I

   (2020) Variables associated with subclinical atherosclerosis among rheumatoid arthritis patients of Gulf Cooperative Council countries. Saudi Med J 41:128–137.

   OpenUrlAbstract/FREE Full Text
2. ↵
   1. Wen T,
   2. Rothenberg ME

   (2016) The regulatory function of eosinophils. Microbiol Spectr 4:1–12.

   OpenUrl
3. ↵
   1. Hannawi S,
   2. Haluska B,
   3. Marwick TH,
   4. Thomas R

   (2007) Atherosclerotic disease is increased in recent-onset rheumatoid arthritis:a critical role for inflammation. Arthritis Res Ther 9:R116.

   OpenUrlCrossRefPubMed
4. 1. Hannawi S,
   2. Marwick TH,
   3. Thomas R

   (2009) Inflammation predicts accelerated brachial arterial wall changes in patients with recent-onset rheumatoid arthritis. Arthritis Res Ther 11:R51.

   OpenUrlCrossRefPubMed
5. ↵
   1. Hannawi S,
   2. Hannawi H,
   3. Alokaily F,
   4. Naredo E,
   5. Moller I,
   6. Al Salmi I

   (2018) Recent-onset of rheumatoid arthritis leads to increase in wall thickness of left anterior descending coronary artery. An evidence of subclinical coronary artery disease. Saudi Med J 39:1213–1217.

   OpenUrlAbstract/FREE Full Text
6. ↵
   1. Marx C,
   2. Novotny J,
   3. Salbeck D,
   4. Zellner KR,
   5. Nicolai L,
   6. Pekayvaz K,
   7. et al.

   (2019) Eosinophil-platelet interactions promote atherosclerosis and stabilize thrombosis with eosinophil extracellular traps. Blood 134:1859–1872.

   OpenUrl
7. ↵
   1. Sundström J,
   2. Söderholm M,
   3. Borné Y,
   4. Nilsson J,
   5. Persson M,
   6. Östling G,
   7. Melander O,
   8. et al.

   (2017) Eosinophil cationic protein, carotid plaque, and incidence of stroke. Stroke 48:2686–2692.

   OpenUrlAbstract/FREE Full Text