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Review ArticleReview Article
Open Access

High mobility group box-1 and cardiovascular diseases

Hua-Sheng Ding and Jun Yang
Saudi Medical Journal May 2010, 31 (5) 486-489;
Hua-Sheng Ding
Department of Cardiology, The First College of Clinical Medical Sciences, China Three Gorges University & Yichang Central People's Hospital, Yichang, The People's Republic of China.
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Jun Yang
Department of Cardiology, The First College of Clinical Medical Sciences, China Three Gorges University & Yichang Central People's Hospital, Yichang, The People's Republic of China.
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Abstract

High mobility group box-1 (HMGB1) is a highly conserved, ubiquitous protein in the nuclei and cytoplasm of almost all cells. After binding to its receptor, HMGB1, which is derived from necrotic cells and activated macrophages/monocytes, functions as a critical mediator of inflammation and promotes tissue repair and regeneration. Many recent studies demonstrated that HMGB1 played a pivotal role in cardiovascular diseases, such as atherosclerosis, myocardial ischemia/reperfusion injury, heart failure, and myocardial infarction. In this review, we focus on HMGB1 and summarize the association of HMGB1 with cardiovascular diseases.

  • Copyright: © Saudi Medical Journal

This is an open-access article distributed under the terms of the Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Saudi Medical Journal: 31 (5)
Saudi Medical Journal
Vol. 31, Issue 5
1 May 2010
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High mobility group box-1 and cardiovascular diseases
Hua-Sheng Ding, Jun Yang
Saudi Medical Journal May 2010, 31 (5) 486-489;

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High mobility group box-1 and cardiovascular diseases
Hua-Sheng Ding, Jun Yang
Saudi Medical Journal May 2010, 31 (5) 486-489;
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© 2025 Saudi Medical Journal Saudi Medical Journal is copyright under the Berne Convention and the International Copyright Convention.  Saudi Medical Journal is an Open Access journal and articles published are distributed under the terms of the Creative Commons Attribution-NonCommercial License (CC BY-NC). Readers may copy, distribute, and display the work for non-commercial purposes with the proper citation of the original work. Electronic ISSN 1658-3175. Print ISSN 0379-5284.

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