Research ArticleOriginal Article
Open Access
Association of CYP2C19, TNF-α, NOD1, NOD2, and PPARγ polymorphisms with peptic ulcer disease enhanced by Helicobacter pylori infection
Laith N. AL-Eitan, Fouad A. Almomani and Sohaib M. Al-Khatib
Saudi Medical Journal January 2021, 42 (1) 21-29; DOI: https://doi.org/10.15537/smj.2021.1.25654
Laith N. AL-Eitan
From the Department of Biotechnology and Genetic Engineering (AL-Eitan, Almomani) Jordan University of Science and Technology and from the Department of Pathology and Microbiology (Al-Khatib), Jordan University of Science and Technology, Irbid, Jordan
MSc, PhDFouad A. Almomani
From the Department of Biotechnology and Genetic Engineering (AL-Eitan, Almomani) Jordan University of Science and Technology and from the Department of Pathology and Microbiology (Al-Khatib), Jordan University of Science and Technology, Irbid, Jordan
MSc, PhDSohaib M. Al-Khatib
From the Department of Biotechnology and Genetic Engineering (AL-Eitan, Almomani) Jordan University of Science and Technology and from the Department of Pathology and Microbiology (Al-Khatib), Jordan University of Science and Technology, Irbid, Jordan
MDReferences
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In this issue
Association of CYP2C19, TNF-α, NOD1, NOD2, and PPARγ polymorphisms with peptic ulcer disease enhanced by Helicobacter pylori infection
Laith N. AL-Eitan, Fouad A. Almomani, Sohaib M. Al-Khatib
Saudi Medical Journal Jan 2021, 42 (1) 21-29; DOI: 10.15537/smj.2021.1.25654
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